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By M. J. Mulvany (auth.), Dr. Giacomo Bruschi, Dr. Alberico Borghetti (eds.)

ISBN-10: 3662009838

ISBN-13: 9783662009833

ISBN-10: 3662009854

ISBN-13: 9783662009857

In the final 20 years, investigations on the mobile point have steadily won floor within the context of high blood pressure study. This collection of process is because of some degree to the building up of information that molecular and mobile biology were generating at a continuing price. because the contents checklist of this quantity exhibits, a wide mass of labor has been directed to gaining a few perception into pathogenetic mechanisms. The pathogenesis of basic high blood pressure has been gradually categorised as a different organic challenge, no longer amenable to the theoretical versions that proved profitable in knowing the character of secondary different types of high blood pressure. whilst, nice efforts were made to simplify this challenge by way of checking out, if attainable, a couple of the most important mechanisms from the community of contributory components within the law of blood strain. the concept what's to be sought is a major structural and/or useful fault in arterial muscle has met with common reputation. The power of this argument lies within the incontrovertible fact that peripheral vascular resistance is elevated in all different types of high blood pressure and, in flip, the diameter of resistance vessels is the dominant consider the computation of overall in line with­ ipheral resistance. at the foundation of this, cardiovascular structural variation used to be proposed as a favorable suggestions mechanism tending to take care of high blood pressure, as soon as all started, regardless of the beginning issue is.

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Atherosclerosis 42:15-20 40 Y. : Cellular Mechanisms of Spontaneous Hypertension and Stroke 45. Yamori Y, Nara Y, Kanbe T, Imafuku H, Mori K, Kihara M, Horie R (1982b) Diversity of membrane abnormalities in spontaneous hypertension. Clin Sci 63 [Suppl 8]:27s-29s Yamori Y, Nara Y, Imafuku H (1984a) Biomembrane abnormalities in spontaneous hypertension. In: Villarreal H, Sambhi MP (eds) Topics in pathophysiology of hypertension. Martinus Nijhoff, Boston, pp 3-13 Yamori Y, 19awa T, Tagami M, Kanbe T, Nara Y, Kihara M, Horie R (1984b) Humoral trophic influence on cardiovascular structural changes in hypertension.

References 1. 2. 3. 4. Aalkjaer C, Heagerty AM, Petersen KK, Swales JD, Mulvany MJ (1987) Evidence for increased media thickness, increased neuronal amine uptake, and depressed excitation - contraction coupling in isolated resistance vessels from essential hypertensives. Circ Res 61(2):181-186 Antonaccio M, Kerwin L (1981) Pre- and post junctional inhibition of vascular sympathetic function by captopril in SHR. Hypertension 3:54-62 Asaad M, Antonaccio M (1982) Vascular wall renin in spontaneously hypertensive rats: potential relevance to hypertension maintenance and antihypertensive effect of captopril.

22. 23. 24. 47 Berk BC, Vekshtein V, Gordon HM, Tsuda T (1989) Angiotensin II-stimulated protein synthesis in cultured vascular smooth muscle cells. Hypertension 13(4): 305-314 Campbell-Boswell M, Robertson A (1981) Effects of angiotensin II and vasopressin on human smooth muscle cells in vitro. Exp Mol Pathrol 35:265-276 Charnley-Campbell J, Campbell G, Ross R (1979) The smooth muscle cell in culture. Physiol Rev 59:1-61 Dzau V (1986) Significance of the vascular renin-angiotensin pathway.

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Cellular Aspects of Hypertension by M. J. Mulvany (auth.), Dr. Giacomo Bruschi, Dr. Alberico Borghetti (eds.)

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